Studying Dementia under the mebolic level.

Introduction

My surname is 'Mikawa' which is the name of this local area in Aichi prefecture, however my family has originated from Chiba.
  My research subject was changed during my reseach experiences. I studied biochemistry about the structure of actin fillament in undergraduate. I was so impressed by the passion of professor and the researchers who taught me, so I am doing research now. Then I went to a graduate course that compared sugar chains between fresh-water and seawater fish. Although there was a most-advanced big lab, I was worrying about the subject and my future. As a result, I have chosen human brain as my subject. Then, I started to study on a cortex-specific genes in primate but failed 4 times of entrance examination for a doctorate course. Now I am in a post of part-time researcher and student in a university.
  I have thought the Alzheimer's disease would be cured in the near future when I come across it in a lecture. However, a lot of difficulties on the treatment. It is one of the most challenging subject that becoming serious not only in Japan but also in the world. Also I am very lucky that I could meet the research specialist of dementia here, the most progressive instutute in the world.
  Dr Takikawa teaches me a good lesson of basic biochemistry as fledgling reseacher. I will study hard to find novel things and to become a good researcher who can contribute to society.

My favorites: Camera, Pub crawl (Craft beer), Cooking, Cat, Rock FES etc.

Subject

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Contact information

Ryuta Mikawa, BSc

Lab Radiation Safety,

National Center for Geriatrics and Gerontology

35 Gengo, Morioka-cho, Obu-City,

Aichi 474-8511, Japan

e-mail:

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Recent publications

Tajima Y, Ishikawa M, Maekawa K, Murayama M, Senoo Y, Nishimaki-Mogami T, Nakanishi H, Ikeda K, Arita M, Taguchi R, Okuno A, ºMikawa R, Niida S, Takikawa O, Saito Y. Lipidomic analysis of brain tissues and plasma in a mouse model expressing mutated human amyloid precursor protein/tau for Alzheimer's disease. Lipids Health Dis. 2013 May 9;12:68. doi: 10.1186/1476-511X-12-68.
[Conference]
ºMikawa R, Okuno A, Yoshimi T, Okada K, Takayanagi A, Takikawa O. Implication of Brain Microvessel Endothelial Cell-derived Beta-Amyloid Peptide in Cerebral Amyloid Angiopathy Associated with Alzheimer's Disease. Alzheimer's Association International Conference (AAIC) 2013, July 14, Boston, USA.
Okuno A, Yoshimi T, Okada K, ºMikawa R, Takayanagi A, Takikawa O. Pathogenic role of IDO in Alzheimer’s disease : Astrocytes but not neurons are responsible for the increase of amyloid β peptide induced by neurotoxin quinolinic acid. ISTRY 2012, November 7, 2012, Sydney, Australia.